How to tell if your child has autism spectrum disorder?

Sep 5, 2021 Contact

In April this year, a team of researchers from the University of Melbourne published an analysis of more than 60,000 scans of the brains of more,000 children from around the world.

The analysis showed that those children with autism spectrum disorders have abnormally high levels of microglia.

The researchers also found that microglial cells, which are part of the immune system, were particularly prominent in autism spectrum children, suggesting that they had a higher risk of developing autism.

This led to a number of hypotheses about why autism spectrum conditions might be more common in children with genetic or environmental factors.

The team found that these genes were linked to the risk of autism, but also a number related to a range of psychiatric illnesses.

The theory goes that autism spectrum symptoms are a consequence of abnormal microgliosis and inflammation, and that these factors increase the risk for developing autism, such as social isolation, depression and obsessive-compulsive symptoms.

The study found that children with autistic spectrum disorders were about four times more likely to have microgliotemporal dementia, or the disease of the brain’s white matter.

The disorder is characterised by a progressive decline in grey matter in the brain, the brain tissue that supports consciousness and cognition.

According to the National Autistic Society, the number of autistic children in Australia has increased from 15,000 in 2005 to 30,000 today.

Autism spectrum disorder symptoms were also significantly linked to mental health issues.

A study published in the journal JAMA Psychiatry in 2015 found that autistic children were more likely than the general population to have major depression, anxiety and anxiety disorder.

And a study published earlier this year in the Lancet Psychiatry found that autism is linked to a high rate of suicidal thoughts.

The paper, published in BMC Psychiatry, compared data from over 200,000 people across the world to the data from children diagnosed with autism from birth to the age of 18.

The data showed that children who had autism spectrum condition were more than five times more than those who had not, and also had a significantly higher rate of anxiety disorders, anxiety related psychotic symptoms, and suicidal thoughts and attempts.

It was also found there was a significant association between autism spectrum and anxiety and depression.

However, researchers from UMRL and UMRB found that the increased risk of suicidal attempts was largely explained by a genetic variation in the gene rs132627 that has been linked to autism.

The gene is a key gene in the development of the nervous system and it regulates several important aspects of the central nervous system.

This gene variant is found in the autism spectrum, but it is thought to be highly common in those with other genetic disorders as well.

Autism is often thought to affect the brain via the central neural pathways.

A recent study in the Journal of Autism and Developmental Disorders, published by UMRM and UMLB, also found increased risk for autism in children whose fathers had autism.

Children who had both autism and autism spectrum spectrum disorder were more closely related to the child than children who were diagnosed with only one autism spectrum diagnosis.

The link between autism and paternal autism was strongest in men, but the link between the two disorders was not found in women.

The UMR team, led by Dr Helen Smith, a researcher in the UMR lab, say this is the first study to look at autism spectrum related risks in men and women.

“In addition to the strong relationship between autism risk and maternal maternal autism, the data also suggest that maternal risk is associated with paternal autism risk as well,” Dr Smith said.

“The link between paternal autism and maternal autism risk is also strong in both men and Women.”

The study was funded by the Australian Research Council (ARC) and the Australian National Health and Medical Research Council.

This article first appeared on the UDR website.

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